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Treatment of hypertension. No pils any more, longlived effect.  

The mechanism by which the primary hypertonia develops is the following: due to stress factors the impulses generated by the vasomotor centers (cortex, diencephalons, medullaHouses Appartments oblongata) to the vegetative systema nervosum – VSN, are with changed parameters (amplitude, frequency or phase). That disturbs the equilibrium between the two parts of VSN – simpaticus and parasimpaticus and as a result the action of the simpatic part starts to predominate. The simpatic activity accelerates, and hence the stimulation of the alpha and Beta receptors in the cardiac muscle is increased. That increases the beat volume and cardiac frequency, which leads to an increase of the peripheral vascular resistance. At a later stage the increase of the peripheral vascular resistance leads to changes in the permeability of the vascular wall, and thus C++ ions get into it (intracellular). Na++ ions accumulate extracellular. The vascular wall thickens and tumefactions may appear. The permeability of the vascular wall may not change. In some cases the permeability changes and in others it doesn’t. At a later stage of the hypertonia’s development another mechanism takes place. The balance between some substances and ferments with pressed and depressed effect gets disturbed in it. The quantity of ferments and substances with pressed effect increases on account of others that depress their activity. This changes the sensibility of the arterial vessels and thus pressed effect is exercised on these substances. This actually is about the rennin- angiotensin - aldosteron system. Angiotensin-2 is a substance that has big vasoconstrictive effect, and the aldosteron is a hormone that takes part in the mineral metabolism. The increase of the aldosteron leads to an accumulation of NaCl in the blood vessels’ wall, and thus it gets thickened and the lumen of the vessels narrows. Besides aldosteron the catecholamines (adrenal hormones) also take part in the system. The neutral lipids and the prostaglandins have depressed (vasodilator) effect. The latter reduce the sensibility of the vascular wall. The prostaglandins are antagonists of angiotensin-2 – substances with vasoconstrictive effect. The prostaglandins influence the intrarenal haemodynamics (the renal blood flow). The hypertonia may develop as a result of the impact of one, two or three factors. The first factor is a disturbance of the vegetative vascular innervation. The second factor is the calcium invasion in the vascular wall and the third factor is nerve-hormonal reasons. The deficit of b-receptors in the vascular walls is of great significance for the development of the primary hypertonia. They give information on their state in the vasomotor centers of the cerebrum. The deficit of b-receptors means lack of information in CNS. This confuses the synchronization of the signals that control the blood pressure and hence CNS no longer controls the systems that control the blood pressure. As a result the balance between the biologically active components in the organism and the antagonism (the balance) between the simpaticus and the parasimpaticus is disturbed. With the method “Impulse therapy” the antagonism between the two parts of the VNS recovers, and thus the simpaticus activity gets neutralized. This normalizes the beat volume and the cardiac frequency, and hence the increased peripheral vascular resistance normalizes. Strengthening of the vascular walls is achieved, with improvement of the metabolism, which recovers the changed permeability. As far as the nerve-hormonal mechanism is concerned an effect is achieved through influencing the hypophysis and the hypothalamus. The level of the aldosteron is regulated, which normalizes the mineral metabolism. On the other hand the production of substances and ferments with depressed (vasodilator) effect is stimulated, so the balance between the substances with vasoconstrictive and vasodilator effect is recovered. With the Impulse therapy the synthesis of receptors in the blood vessels is stimulated. The time needed for recovery of the normal blood pressure for the lighter stages of hypertonia is between 6 and 8 months, and for the ones that are more serious - about 12 months. Meantime the quantity of b-receptors increases. After this period the medicines for hypertonia stop being taken.

The hypertension is one of the most frequantly met disease. There are a lot of theories for the reasons for its arising. Neither of them has succeeded to explain and prove the concrete mechanism for these disease. For that reason its medication is symptomatic and medicaments for hypertension are taken constantly till the end of life.
Sensationaly but this disiese can be completely cured. The causes of high blood pressure is due to hypofunction of the thyroid gland. This revolutionary conclusion is proved by the method applied for healing the thyroid gland. Normalizing the synthesis of necessery thyroid hormons for the organizm the values of RR also get normalized. This normalization is possible when the brain center recover the full control over the thyroid gland. With the method developed by the bulgarian specialists the hypertension normalise within 6 months. Patients are stoping to take any medicaments. Long-lived of the disease as well as the values of the RR has no significance. The attendant hypertonic palpitation also disapear. This method is tested more then 20 years and can be cured patiens only with essential hypertention. If the patien has kidney problems the  terrapy  can not help. High blood presure is the first forerunner of heart attacks and brain insults.  Low RR is equaly dangerous as well as the high one. Fortunately allready there is a solution to this medical problem.
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